DKA
Across
- 5. Critically ill patients with severe hyperglycemia resulting from DKA should be treated immediately with a bolus of (2 words)
- 7. "The Five I's" in the differential diagnosis for the cause of hyperglycemic crisis include infarction, infant (pregnancy), indiscretion (drug ingestion), insulin lack (nonadherence or inappropriate dosing), and ________
- 9. This guy (last name) described the deep and labored breathing seen in DKA
- 10. Symptoms of DKA include vomiting, __________, deep gasping breathing, confusion and occasionally coma.
- 12. A ketotic odor on a DKA patients breath is often described as _________
- 13. This counterregulatory hormone increases during DKA
- 14. ________ function may be impaired in DKA as a result of dehydration
- 15. It sounds like a good idea, but studies have shown that IV __________ should only be used in patients with severe acidosis (pH < 6.9) (Hint: The resident suggested giving this in our case and the attending was annoyed)
- 17. Insulin can decrease __________ levels in the blood and cause dangerous irregularities in the heart rate
- 18. This painful "draw" is usually performed to demonstrate acidosis in DKA (acronym)
Down
- 1. DKA has an annual cost of about $2.4 ________ in the united states, up to half of the total cost of caring for people with type I diabetes
- 2. DKA is distinguished from other diabetic emergencies by the presence of large amounts of __________ in blood and urine.
- 3. The American Diabetes Association has categorized DKA as mild, moderate, and severe. _______ can only occur in the severe category
- 4. Young patients with recurrent episodes of DKA may have an underlying __________ (2 words, hint: There was a lecture about this yesterday)
- 6. Though rare in adults, _______ edema occurs in .3-1% of children with DKA
- 8. The absence of insulin leads to the release of ________ (2 words) from adipose tissue
- 11. DKA is caused by a shortage of ________
- 16. A 1998 study published in the Archives of Internal Medicine determined that this illegal drug's use was a risk factor for diabetic ketoacidosis, either because of its effects on counter-regulatory hormones or because of the individual’s subsequent tendency to omit insulin doses.