Neuropathology Terms and Diseases

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Across
  1. 2. Degeneration of this structure in non-chondrodysplastic dogs is the inciting cause for Type II IVDD
  2. 7. Diagnosis can be confirmed by performing IHC and Western-Blot for this prion disease in the obex, tonsils, and retropharyngeal lymph nodes
  3. 10. Potential in utero cause of cerebellar hypoplasia in cats
  4. 11. _________________ can be caused by congenital or acquired portosystemic shunts
  5. 15. Potentially toxic to the outer hair cells of the inner ear
  6. 18. Deficiency responsible for Night Blindness
  7. 21. Commonly seen in dogs with Diabetes Mellitus but not cats
  8. 22. Listeria spp. are often found in incompletely fermented _____________
  9. 24. Edema present here is pathognomonic for edema disease in pigs
  10. 30. Tubular cavitation in the spinal cord. May communicate with the central canal but isn't lined by ependymal cells
  11. 32. Inflammation of the iris and ciliary body
  12. 33. ______________ deficiency can cause polioencephalomalacia in ruminants
  13. 36. Caused by E. coli shiga-toxin
  14. 37. Due to a malformation in the filtration angle of the anterior chamber of the eye (Goniodysgenesis)
  15. 39. Inflammation of the Choroid and Retina
  16. 42. Amyloid deposition in the non-pigmented region of the ciliary body is pathognomonic for this disease in horses
  17. 44. Associated lesion of Thrombotic Meningoencephalitis
  18. 45. Deficiency responsible for Feline Central Retinal Degeneration (FCRD). Also associated with Dilatative Cardiomyopathy (DCM)
  19. 46. Accumulation of cerebrospinal fluid within the ventricular system
  20. 47. Inversion of the eyelid margin
  21. 48. Cause of Equine Protozoal Myelitis
  22. 49. Listeria reaches the brain via the Facial Nerve and this other nerve
  23. 50. Can cause the infiltration of perivascular eosinophils in the brain of pigs
  24. 51. Defective closure of the neural tube resulting in cyclopia/synophthalmus
Down
  1. 1. Route by which nasopharyngeal polyps migrate in cats
  2. 3. Portion of the spinal cord most commonly affected in West Nile Virus infections
  3. 4. Causes nigropallidal encephalomalacia in horses
  4. 5. Receptor (or the lack thereof) responsible for the pathogenesis of colonic aganglionosis
  5. 6. Found in moldy corn. Causes leukoencephalomalacia in horses
  6. 8. Cholesteatoma or keratinizing cysts that arise from the tympanic membrane
  7. 9. Neurotoxicity with this substance can damage vascular endothelial cells of the brain and result in congestion, cell swelling, and necrosis. Potential intranuclear, acid-fast inclusion bodies
  8. 12. Inflammation of the lens
  9. 13. Fluoresces under UV light
  10. 14. Can result from in utero exposure to Griseofulvin in cats
  11. 16. Inflammation of the salivary gland. Occurs in infection with Rabies Virus
  12. 17. Otherwise known as Vogt-Koyanagi-Harada-like Syndrome. Cell-mediated autoimmunity against melanocytes in the uvea, retina, and skin
  13. 19. Should be a rule-out for any neurologic case
  14. 20. Occurs as an acute, generalized disease in birds. Horses and humans are atypical, dead-end hosts
  15. 23. Nerve by which virus particles travel from the CNS to the salivary gland in infections with Rabies Virus
  16. 25. Region of spinal cord most commonly affected by Equine Protozoal Myelitis
  17. 26. Intracytoplasmic inclusion bodies in the cerebellum associated with Rabies Virus
  18. 27. Should be a primary rule-out when considering Horner's Syndrome
  19. 28. Possible sequela of Dysautonomia. Can result in aspiration pneumonia
  20. 29. Inflammation of the middle ear
  21. 31. Proteinaceous Contagion (i.e. Bovine Spongiform Encephalopathy)
  22. 34. Cause of Thrombotic Meningoencephalitis in cattle
  23. 35. Degeneration of this structure in chondrodysplastic dogs is the inciting cause for Type I IVDD
  24. 38. Inflammation of the Lens, Retina, and Choroid
  25. 40. Occurs when there is damage to the Ciliary Body
  26. 41. Accumulation of cerebrospinal fluid following primary destruction of white matter
  27. 43. Has both microabscesses (suppurative component) and lymphohistiocytic perivascular cuffing (nonsuppurative component)