Pain (Part 2): Practice OCAT #4

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Across
  1. 2. Modern framework integrating biology psychology and environment in pain.
  2. 6. Major ascending pathway carrying pain and temperature.
  3. 7. Loss of inhibitory control increasing dorsal horn excitability.
  4. 8. Descending or local alteration of pain signal strength.
  5. 10. Organization that formally defines pain in modern medicine.
  6. 11. Fast excitatory receptor mediating early synaptic transmission.
  7. 17. Increased responsiveness of neurons after injury.
  8. 18. Endogenous system that inhibits nociceptive transmission.
  9. 20. Primary excitatory neurotransmitter in pain pathways.
  10. 21. Primary sensory neuron specialized for detecting tissue damage.
  11. 22. Brain to spinal cord pathway that can suppress pain.
  12. 25. Major inhibitory neurotransmitter limiting dorsal horn excitation.
Down
  1. 1. Pain evoked by a normally nonpainful stimulus.
  2. 3. Endogenous peptide activating opioid receptors.
  3. 4. Activity dependent strengthening underlying persistent pain states.
  4. 5. Relay station directing nociceptive input to cortex.
  5. 9. Ascending movement of nociceptive signals to the brain.
  6. 12. Protective short duration pain following injury.
  7. 13. Receptor central to synaptic plasticity and chronic pain development.
  8. 14. Pain state persisting beyond normal tissue healing.
  9. 15. Conversion of noxious stimulus into electrical activity.
  10. 16. Conscious experience generated after cortical processing.
  11. 19. Exaggerated response to a normally painful stimulus.
  12. 23. Site where pain becomes a subjective experience.
  13. 24. Peripheral biological response that lowers nociceptor threshold.